Actinodinium apsteini
Diagnosis
Diagnosis_Genus: Actinodinium Chatton & Hovasse 1937. Parasite extended (and surrounded) by spikes (rays) which are expansion of the cytoplasm and formed cuticle. This parasite develops in the wall of the stomach, without penetration inside the intestine, but can penetrate the nervous system and the digestive gland. The parasite may be surrounded by several cuticles. The nucleus is central, without condensed chromosomes during the trophont stage. At the end of the trophont maturation, one spike (ray) perforates the intestine membrane, and the parasite flows into the intestine, leaving the external membrane of the parasite inside the host. Sporogenesis and spores not observed.
Diagnosis_Species: Actinodinium apsteini Chatton & Hovasse 1937. Parasite of Acartia clausi, located in the anterior dorsal region of the cephalothoracique cavity, generally in contact of the digestive tube. Infections are not lethal for its host, the parasite does not castrate its host. Sporogenesis and spores not observed.
Body_trophont_length: 40-125 µm
Etymology
apsteini: in honor of Pr Apstein, who first described this species.
Type species
This is the type species of the genus.
Type illustration / Type locality / Type specimen
Type locality: Roscoff (France)
Type host: Acartia clausi
Ecology
Substrate: endozoic
Sociability_trophont: solitary
Salinity: marine
Life cycle
Generation: <1 month
Reproduction_mode: asexual
Symbiont: horizontal
Feeding behaviour
Reference(s)
Observation site(s)
HOSTS
Association with... | Region origin | Name of site | In reference... |
---|---|---|---|
Acartia clausi |
Actinodinium apsteini, n. g., n. sp., peridinien parasite entérocoelomique des Arcartia (copépodes). Archives de Zoologie Expérimentale et Générale 79:24-29. (1938) |
||
Calanus finmarchicus | Kattegat |
Parasiten von Calanus finmarchicus. Wiss. Meers. Unters. Abt. Kiel, Neue Folg. XIII:207-223. (1911) |